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Ageing
Simple Ways To Slow Down The Ageing Process
Date: 17/02/05
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Today we'll look at another cell function that impacts the ageing process. Mitochondria are cell organelles (literally; organs of the cell) that serve as power plants, processing nutrients and oxygen that feed the cells. But toxins and diseases weaken mitochondria, often reducing cellular efficiency, as we grow older. Fortunately there are simple steps we can take to prevent the breakdown and loss of vitally important mitochondria...In yesterdays e-alert, I told you about a Duke University study in the US that demonstrates how cells ration iron, first making sure that the most essential cell functions get their share, and then depriving certain genes of iron if the iron supply is insufficient.
Today well look at another cell function that impacts the ageing process.
Mitochondria are cell organelles (literally; organs of the cell) that serve as power plants, processing nutrients and oxygen that feed the cells. But toxins and diseases weaken mitochondria, often reducing cellular efficiency, as we grow older. The results range from the typical signs of ageing to chronic and debilitating illnesses.
Fortunately there are simple steps we can take to prevent the breakdown and loss of vitally important mitochondria.
The diabetes connection
Disorders sometimes associated with mitochondrial dysfunction include chronic fatigue syndrome, dementia, cardiomyopathy (heart muscle disease), and kidney ailments. To that list we can now add type 2 diabetes.
Knowing that the accumulation of fat in liver and muscle tissue is an accurate predictor of insulin resistance (a precursor of type 2 diabetes), a team of scientists at Yale University School of Medicine in the US used non-invasive scans to examine the muscles and livers of 29 people. Subjects were made up of two groups: 13 were between the ages of 18 and 39, and 16 were between the ages of 61 to 84. All subjects were healthy and showed no signs of pre-diabetic conditions such as excessive body weight.
Results showed that the group of older subjects not only had a higher accumulation of fat in their liver and muscle tissue than the younger group, but also an average of 40 percent lower metabolic activity of the mitochondria. This is significant because one of the key jobs of mitochondria is to convert both fatty acids and glucose into energy.
The Yale researchers conclusion - that a lack of the mitochondria necessary to burn fat promotes a lowered sensitivity to insulin - leads to this practical advice: Get up and exercise. Because studies have shown that regular physical activity stimulates and even increases the amount of mitochondria within your cells.
It takes two
In addition to physical activity, there are supplements that can help keep the mitochondria plentiful and lively.
For instance, many of the benefits of the amino acid acetyl L-carnitine (ALC) - such as the protection of brain nerve cells from degeneration - are attributed to its stimulating effects on the mitochondria.
Unfortunately, some research has shown that ALC can increase oxidative stress. But a study from the University of California in the US found that combining ALC with alpha lipoic acid (ALA) not only eliminates the concerns about oxidative stress, but also magnifies ALCs anti-ageing effects.
The results of that study suggest that ALC and ALA supplements support the health of mitochondria, resulting in a boost to the metabolism that may even improve memory while fighting oxidative stress. The usual recommended dosage for ALC is 250 mg to 2 grams daily, and 100 to 600 mg of ALA. Both supplements are available through vitamin suppliers and health food stores in the UK.
Back to topToday well look at another cell function that impacts the ageing process.
Mitochondria are cell organelles (literally; organs of the cell) that serve as power plants, processing nutrients and oxygen that feed the cells. But toxins and diseases weaken mitochondria, often reducing cellular efficiency, as we grow older. The results range from the typical signs of ageing to chronic and debilitating illnesses.
Fortunately there are simple steps we can take to prevent the breakdown and loss of vitally important mitochondria.
The diabetes connection
Disorders sometimes associated with mitochondrial dysfunction include chronic fatigue syndrome, dementia, cardiomyopathy (heart muscle disease), and kidney ailments. To that list we can now add type 2 diabetes.
Knowing that the accumulation of fat in liver and muscle tissue is an accurate predictor of insulin resistance (a precursor of type 2 diabetes), a team of scientists at Yale University School of Medicine in the US used non-invasive scans to examine the muscles and livers of 29 people. Subjects were made up of two groups: 13 were between the ages of 18 and 39, and 16 were between the ages of 61 to 84. All subjects were healthy and showed no signs of pre-diabetic conditions such as excessive body weight.
Results showed that the group of older subjects not only had a higher accumulation of fat in their liver and muscle tissue than the younger group, but also an average of 40 percent lower metabolic activity of the mitochondria. This is significant because one of the key jobs of mitochondria is to convert both fatty acids and glucose into energy.
The Yale researchers conclusion - that a lack of the mitochondria necessary to burn fat promotes a lowered sensitivity to insulin - leads to this practical advice: Get up and exercise. Because studies have shown that regular physical activity stimulates and even increases the amount of mitochondria within your cells.
It takes two
In addition to physical activity, there are supplements that can help keep the mitochondria plentiful and lively.
For instance, many of the benefits of the amino acid acetyl L-carnitine (ALC) - such as the protection of brain nerve cells from degeneration - are attributed to its stimulating effects on the mitochondria.
Unfortunately, some research has shown that ALC can increase oxidative stress. But a study from the University of California in the US found that combining ALC with alpha lipoic acid (ALA) not only eliminates the concerns about oxidative stress, but also magnifies ALCs anti-ageing effects.
The results of that study suggest that ALC and ALA supplements support the health of mitochondria, resulting in a boost to the metabolism that may even improve memory while fighting oxidative stress. The usual recommended dosage for ALC is 250 mg to 2 grams daily, and 100 to 600 mg of ALA. Both supplements are available through vitamin suppliers and health food stores in the UK.
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